These results demonstrate that it is the adhesin-CEACAM interaction, which serves as the trigger for increased extracellular matrix adhesion of the infected cells
These results demonstrate that it is the adhesin-CEACAM interaction, which serves as the trigger for increased extracellular matrix adhesion of the infected cells. Previously, CEACAM engagement by bacteria has been shown to induce CD105 expression in epithelial Rabbit polyclonal to Acinus cells, which was a pre-requisite for enhanced matrix adhesion of infected cells [16]. ME-180…